Das Klinikum

Dr. med. Bernhard N. Bohnert

Dr. med. Bernhard N. Bohnert

Funktionsoberarzt

Sektion für Nieren- und Hochdruckkrankheiten

Facharzt für Innere Medizin und Nephrologie

Kontakt

Telefonnummer: 07071 29-83172

E-Mail-Adresse: bernhard.bohnert@med.uni-tuebingen.de

Klinik / Institut / Zentrum

  • Bohnert BN, Menacher M, Janessa A, Wörn M, Schork A, Daiminger S, Kalbacher H, Häring HU, Daniel C, Amann K, Sure F, Bertog M, Haerteis S, Korbmacher C, Artunc F. Aprotinin prevents proteolytic epithelial sodium channel (ENaC) activation an volume retention in nephrotic syndrome. Kidney Int. 2018 Jan; 93(1):159-172.
  • Bissinger R, Nemkov T, D'Alessandro A, Grau M, Dietz T, Bohnert BN, Essigke D, Wörn M, Schaefer L, Xiao M, Beirne JM, Kalo MZ, Schork A, Bakchoul T, Omage K, Kong L, Gonzalez-Menendez I, Quintanilla-Martinez L, Fehrenbacher B, Schaller M, Dhariwal A, Birkenfeld AL, Grahammer F, Qadri SM, Artunc F. Proteinuric chronic kidney disease is associated with altered red blood cell lifespan, deformability and metabolism. Kidney Int. 2021 Dec;100(6):1227-1239. doi: 10.1016/j.kint.2021.08.024.
  • Bohnert BN, Essigke D, Janessa A, Schneider JC, Wörn M, Kalo MZ, Xiao M, Kong L, Omage K, Hennenlotter J, Amend B, Birkenfeld AL, Artunc F. Experimental nephrotic syndrome leads to proteolytic activation of the epithelial Na + channel in the mouse kidney. Am J Physiol Renal Physiol. 2021 Oct 1;321(4):F480-F493. doi: 10.1152/ajprenal.00199.2021.
  • Bohnert BN, Daiminger S, Worn M, Sure F, Staudner T, Ilyaskin AV, Batbouta F, Janessa A, Schneider JC, Essigke D, Kanse S, Haerteis S, Korbmacher C, Artunc F. Urokinase-type plasminogen activator (uPA) is not essential for epithelial sodium channel (ENaC)-mediated sodium retention in experimental nephrotic syndrome. Acta Physiol (Oxf). 2019: e13286. 2019/04/22. doi: 10.1111/apha.13286.
  • Xiao M, Bohnert BN, Aypek H, Kretz O, Grahammer F, Aukschun U, Wörn M, Janessa A, Essigke D, Daniel C, Amann K, Huber TB, Plow EF, Birkenfeld AL, Artunc F. Plasminogen deficiency does not prevent sodium retention in a genetic mouse model of experimental nephrotic syndrome. Acta Physiol (Oxf). 2021 Jan;231(1):e13512.
  • Bohnert BN, Artunc F. Induction of Nephrotic Syndrome in Mice be Retrobulbar Injection of Doxorubicin and Prevention of Volume Rretention by Sustained Release Aprotinin. J. Vis. Exp. (135), e57642.
  • Bohnert BN, Gonzalez-Menendez I, Dörffel T, Schneider JC, Xiao M, Janessa A, Kalo MZ, Fehrenbacher B, Schaller M, Casadei N, Amann K, Daniel C, Birkenfeld AL, Grahammer F, Izem L, Plow EF, Quintanilla-Martinez L, Artunc F. Essential role of DNA-PKcs and plasminogen for the development of doxorubicin-induced glomerular injury in mice. Dis Model Mech. 2021 Sep 1;14(9):dmm049038. doi: 10.1242/dmm.049038.
  • Schork A, Bohnert BN, Heyne N, Birkenfeld AL, Artunc F. Overhydration Measured by Bioimpedance Spectroscopy and Urinary Serine Protease Activity Are Risk Factors for Progression of Chronic Kidney Disease. Kidney Blood Press Res. 2020;45(6):955-968.
  • Bohnert BN, Büttner-Herold M, Amann K, Schork A, Birkenfeld AL, Guthoff M, Heyne N, Nadalin S, Artunc F. The Case | A 74-year-old liver transplant recipient with nephrotic-range proteinuria. Kidney Int. 2021 Apr;99(4):1031-1032. doi: 10.1016/j.kint.2020.10.029.
  • Uzun G, Bohnert BN, Althaus K, Nann D, Nadalin S, Heyne N, Fend F, Haap M, Bakchoul T. Organ Donation From a Brain Dead Donor With Vaccine-induced Immune Thrombotic Thrombocytopenia After Ad26.COV2.S: The Risk of Organ Microthrombi. Transplantation. 2022 Mar 1;106(3):e178-e180. doi: 10.1097/TP.0000000000004039.
  • Artunc F, Wörn M, Schork A, Bohnert BN. Proteasuria-The impact of active urinary proteases on sodium retention in nephrotic syndrome. Acta Physiol (Oxf). 2018 Dec 31:e13249.
  • Xiao M, Bohnert BN, Grahammer F, Artunc F. Rodent models to study sodium retention in experimental nephrotic syndrome. Acta Physiol (Oxf). 2022 May 15:e13844. doi: 10.1111/apha.13844.
  • Publikationen